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atac seq high throughput data  (Azenta)

 
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    Azenta atac seq high throughput data
    (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes <t>from</t> <t>ATAC-seq</t> and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).
    Atac Seq High Throughput Data, supplied by Azenta, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/atac seq high throughput data/product/Azenta
    Average 86 stars, based on 1 article reviews
    atac seq high throughput data - by Bioz Stars, 2026-05
    86/100 stars

    Images

    1) Product Images from "MNS induces antiviral protection and suppresses inflammation"

    Article Title: MNS induces antiviral protection and suppresses inflammation

    Journal: bioRxiv

    doi: 10.64898/2026.02.11.705318

    (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes from ATAC-seq and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).
    Figure Legend Snippet: (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes from ATAC-seq and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).

    Techniques Used: Gene Expression, Binding Assay, RNA Sequencing



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    atac seq high throughput data  (Azenta)
    86
    Azenta atac seq high throughput data
    (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes <t>from</t> <t>ATAC-seq</t> and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).
    Atac Seq High Throughput Data, supplied by Azenta, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/atac seq high throughput data/product/Azenta
    Average 86 stars, based on 1 article reviews
    atac seq high throughput data - by Bioz Stars, 2026-05
    86/100 stars
    		  Buy from Supplier

    Image Search Results


    (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes from ATAC-seq and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).

    Journal: bioRxiv

    Article Title: MNS induces antiviral protection and suppresses inflammation

    doi: 10.64898/2026.02.11.705318

    Figure Lengend Snippet: (A) Heatmap of inflammatory pathway gene expression in cells treated with DMSO, LPS, LPS+MNS, or MNS alone, highlighting MNS-mediated regulation relative to LPS. (B) Volcano plot showed DEGs between the treatment of LPS+MNS and LPS alone in RAW264.7 cells. (C) Gene Ontology (GO) enrichment of DEGs in (B). (D) KEGG enrichment of DEGs in (B). (E) Heatmap showed that MNS markedly changed the chromatin accessibility induced by LPS. (F) Heatmap illustrated the differentially accessible regions (DARs), following treatment with DMSO, LPS, LPS-MNS and MNS alone. (G) Distribution of peaks and transcription factor-binding loci relative to transcription start sites (TSS) in the LPS, LPS-MNS, and MNS groups compared to the DMSO group, respectively, as annotated using the ChIPseeker R package. (H) Scatter plot analysis comparing fold changes from ATAC-seq and RNA-seq. (I) Overlap downregulated genes in ATAC-seq and bulk RNA-seq between LPS and LPS-MNS groups in RAW 264.7 cells. (J) KEGG enrichment of overlap genes in (I).

    Article Snippet: For ATAC-seq high-throughput data generated by GENEWIZ (Suzhou, China), raw FASTQ files were first subjected to quality control and adapter trimming with Trim Galore (v0.6.4).

    Techniques: Gene Expression, Binding Assay, RNA Sequencing